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Classification of pulpitis
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I. Acute pulpitis.
1. Hyperemia of pulp
2. Acute localized serous (partial) pulpitis
3. Acute diffused serous pulpitis
4. Acute purulent pulpitis
5. Acute traumatic pulpitis
II. Chronic pulpitis.
1. Chronic fibrous pulpitis
2. Chronic hypertrophic pulpitis (pulp polyp)
3. Chronic gangrenous pulpitis
4. Chronic calcified pulpitis (pulp stone)
III. Exacerbation of chronic pulpitis.
IV. Pulpitis, complicated by apical periodontitis.
| Dental caries is by far the most common cause and is usually obvious unless it has extended under the edge of a restoration. The pulp is involved in the inflammation process due to progression of carious lesion. | Exposure during cavity preparation allows bacteria to enter the pulp and also damages it mechanically. Fracture may either open the pulp chamber or leave so thin a covering of dentine that bacteria can enter. A tooth, particularly a restored premolar, may split, usually under masticatory stress. These small cracks are often invisible, but allow bacteria into the pulp chamber. |
| Some restorations without a protective lining (chemical cured composite that is placed without cement lining), particularly, in the past were sufficiently irritant to kill the pulp. | Over-rapid cavity cutting, especially of deep cavities, can cause immediate damage to the pulp. Large unlined metal restorations may also allow continuous low-grade thermal stimuli to damage the pulp over a longer period. |
